Friday, November 03, 2006

Putative contributors to the secular increase in obesity: exploring the roads less traveled

I was just reading a review published in the November issue of the International Journal of Obesity and thought I'd share, the paper is titled, Putative contributors to the secular increase in obesity: exploring the roads less traveled (full paper) .

The reason I found it interesting was because it explores other possible contributions to our obesity epidemic other then just the big two, reduced physical activity and food marketing practices.

What the paper does is explore ten putative additional explanations though not conclusive, which may contribute to our current obesity crisis. The ten explanations cover:

  1. Sleep debt: Evidence that less sleep can cause increased body weight and that average sleep debt has increased.
  2. Endocrine disruptors: Evidence that endocrine disruptors can increase adiposity and that our exposure has also increased.
  3. Reduction in variability in ambient temperature: Evidence that remaining in the thermoneutral zone promotes adiposity and our time in thermoneutral zones has increased.
  4. Decreased smoking: Evidence that smoking reduces weight and that smoking rates have decreased.
  5. Pharmaceutical iatrogenesis: Evidence that certain pharmaceuticals increase weight and usage has increased.
  6. Changes in distribution of ethnicity and age: Evidence that some age and ethnic groups have higher prevalence of obesity than others and they have increased in relative frequency.
  7. Increasing gravida age: Evidence that greater gravida age increases risk of offspring obesity.
  8. Intrauterine and intergenerational effects: Influences on obesity in utero and even as far back as two generations when oocytes are formed in the grandmother.
  9. Greater BMI is associated with greater reproductive fitness yielding selection for obesity-predisposing genotypes: Proposition that individuals with a genetic predisposition for a higher BMI reproduce at a higher rate then individuals who are genetically predisposed to a lower BMI.
  10. Assortative mating and floor effects: Humans assortatively mate for adiposity.
They also mention but don't go into, other factors such as reduction in breastfeeding, childhood depression, hormones in agricultural species, shift work, reduced calcium or dairy consumption etc.

Anyway I thought some of these explanations were interesting for example a quote from their explanation on endocrine disruptors:

Evidence that endocrine disruptors exposure has increased

Endocrine disruptors have increased in the food chain.39, 40 One example indicator is that polybrominated diphenyl ether concentration in Swedish women's breast milk almost doubled every 5 years from 1972 to 1998.39"

Anyway have a read and let me know what you think...


FAT BITCH said...

Hi Sherrie :)

Thank you for this. I just learned something new and my brain (and diet journey) is better off for it for sure!

Sherrie said...

Hehe your very welcome! :)